据伦敦媒体报道，英国最新的医学研究发现，长痣有意想不到的好处，身上痣愈多的人，更能留住青春的脚步，甚至更长寿。

伦敦大学国王学院的科学家对1800多对双胞胎进行比较研究，发现身上痣愈多的人（超过100颗以上），体内的端粒比身上的痣不超过25颗的人要长。

端粒是染色体末端的结构，随着细胞老化和失去分裂能力，端粒会逐渐缩短长度，换言之，端粒愈长显示细胞老化愈慢。

国王学院双胞胎研究中心巴泰尔博士指出，研究结果首次发现，痣多的人罹患皮肤癌的风险可能高一些，但是他们老化得慢，而且较不易罹患与老化相关的疾病，例如心血管疾病、骨质疏松症等。

报告指出，孩提时长的痣到中年后会慢慢消失，一般白种人平均有30颗痣，有人全身甚至有高达400颗痣，造成这些差异的原因及痣的功能究竟何在，目前仍不明。过去有研究指出，痣的多寡和遗传有关。

依据痣随着年纪增长而消失的现象，巴泰尔博士与研究人员以7年的时间，每年观察被研究的双胞胎染色体端粒长度。结果显示，痣愈多的人，端粒也愈长，同时痣可以维持更久；而端粒短的人不仅痣少，而且随着年龄增长，痣也跟着消失。

不知道痣的多少和人体的运动能力有没有关系？

我还弄不清楚什么是痣呢

Collins等（2003）研究表明，慢性运动疲劳会引起骨骼肌端粒显著缩短。

可以推论，痣多的人，不但运动能力强，而且运动寿命长。当然，这有待调查。

参考文献

Collins M, Renault V, Grobler LA, St Clair Gibson A, Lambert MI, Wayne Derman E, Butler-Browne GS, Noakes TD, Mouly V. 2003. Athletes with exercise-associated fatigue have abnormally short muscle DNA telomeres. Med Sci Sports Exerc. 2003 Sep;35(9):1524-8. INTRODUCTION/PURPOSE: Although the beneficial health effects of regular moderate exercise are well established, there is substantial evidence that the heavy training and racing carried out by endurance athletes can cause skeletal muscle damage. This damage is repaired by satellite cells that can undergo a finite number of cell divisions. In this study, we have compared a marker of skeletal muscle regeneration of athletes with exercise-associated chronic fatigue, a condition labeled the "fatigued athlete myopathic syndrome" (FAMS), with healthy asymptomatic age- and mileage-matched control endurance athletes. METHODS: Muscle biopsies of the vastus lateralis were obtained from 13 patients diagnosed with FAMS and from 13 healthy control subjects. DNA was extracted from the muscle samples and their telomeric restriction fragment (TRF) or telomere lengths were measured by Southern blot analysis. RESULTS: All 13 symptomatic athletes reported a progressive decline in athletic performance, decreased ability to tolerate high mileage training, and excessive muscular fatigue during exercise. The minimum value of TRF lengths (4.0 +/- 1.8 kb) measured on the DNA from vastus lateralis biopsies from these athletes were significantly shorter than those from 13 age- and mileage-matched control athletes (5.4 +/- 0.6 kb, P < 0.05). Three of the FAMS patients had extremely short telomeres (1.0 +/- 0.3 kb). The minimum TRF lengths of the remaining 10 symptomatic athletes (4.9 +/- 0.5 kb, P < 0.05) were also significantly shorter that those of the control athletes. CONCLUSION: These findings suggest that skeletal muscle from symptomatic athletes with FAMS show extensive regeneration which most probably results from more frequent bouts of satellite cell proliferation in response to recurrent training- and racing-induced muscle injury.

谢谢刘老师的解答！